The c-Jun N-terminal kinase (JNK) pathway is activated in human interstitial cystitis (IC) and rat protamine sulfate induced cystitis
In vivo 1H NMR spectroscopy of rat brain at 1 ms echo time
The development of tumor treatment strategies targeting GSH-degrading enzymes faces challenges, including different roles of these enzymes in different types of tumor and their impact on the immune response or tumor microenvironment
MYC drives human hepatocarcinogenesis 16, is frequently amplified or overexpressed in human hepatocellular carcinoma (HCC), and is associated with poorly differentiated tumors and poor liver cancer prognosis 1721
[Cited for in vitro mechanistic context only.] Yue, X., Liu, S.-L., Guo, J.-N., Meng, T.-G., Zhang, X.-R., Li, H.-X., Song, C.-Y., Wang, Z.-B., Schatten, H., Sun, Q.-Y., & Guo, X.-P
Furthermore, miRNA-based therapeutic interventions, leveraging tools like quantitative reverse transcriptase PCR (qRT-PCR), droplet-based digital PCR (ddPCR), and microarrays for profiling, hold revolutionary potential to modulate key pathological cascades, and ultimately enable precision management strategies for early intervention and prevention of DR progression
